Over-expression of JAZF1 reduces proinflammatory cytokines release via inhibition of stress kinases

来源 :第九届湘雅国际糖尿病免疫学论坛 | 被引量 : 0次 | 上传用户:walkeronmoon
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Objective It is now recognized that NAFLD is an inflammatory state associated with increased levels of proinflammatory cytokines and chemokines in the circulation and in the liver.As an inhibitor of the TAK1/TR4 signaling pathway, Juxtaposed with another zinc finger gene 1(JAZF1) has been shown to be involved in gluconeogenesis, lipid metabolism and insulin sensitivity.However, its role in hepatic lipogenesis and chronic low-grade inflammation leading to Non-alcoholic fatty liver disease (NAFLD) remains unknown.The aim of this study was to examine whether JAZF1 overexpression in vivo or in vitro can protect against palmitic acid (PA)-and high fat diet (HFD)-induced systemic inflammatory responses and to explore potential mechanism during this process.
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