论文部分内容阅读
We previously demonstrated that conventional protein kinase C (cPKC)γ was involved in hypoxic preconditioning (HPC)-induced neuroprotection against ischemic injuries of mice with middle cerebral artery occlusion (MCAO)-induced ischemic stroke.We also found that cPKCγ might interact with ubiquitin carboxyl-terminal hydrolase L1 (UCHL1) in the brain of HPC mice by using techniques of functional proteomics.