论文部分内容阅读
Background Intermittent hypoxia (IH), as a major characteristic of obstructive sleep apnea, may represent a chronic intermittent stress, leading to dysregulation of hypothalamic-pituitary-adrenal (HPA) axis, which could contribute to or exacerbate other pathophysiological processes.The aim of this study was to investigate the ultrastructure and function of HPA axis in rats exposure to IH and the intervention of reduced glutathione (GSH).