Foot shock potentiates blood coagulation system of rats

来源 :中国生理学会第24届全国会员代表大会暨生理学学术大会 | 被引量 : 0次 | 上传用户：szscan

【摘要】

：

【作者】

：

程玉文孙珮雯朱晨洁张国兴

【机构】

：

苏州大学医学部基础医学与生物科学学院神经与生理学系,苏州,215123

【出处】

：

中国生理学会第24届全国会员代表大会暨生理学学术大会

【发表日期】

：

2014年7期

【关键词】

：

Coagulation function Foot shock Oxidative stress Renal sympathetic nerve system

下载到本地 , 更方便阅读

下载此文赞助VIP

声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架

论文部分内容阅读

　　[Objective]To observe the influence of foot shock on blood coagulation system of rats.[Methods]Male Sprague Dawley(SD)rats were randomly divided into four groups: Control group(n=6),foot shock group(n=6),renal sympathetic nerve denervation shock group plus foot shock(n=6)and TEMPOL plus foot shock group(n=6).

其他文献

The regulation of phosphorylation of large-conductance,calcium-activated potassium channels in a rat

Background:Trigeminal neuralgia is a disorder of paroxysmal and severely disabling facial pain and continues to be a real therapeutic challenge.At present there are few effective drugs.Here the aim of

会议

BKCa channeltrigeminal neuropathic painchronic constriction injury (CCI)ERKp

Magnesium L-threonate attenuate vincristine-induced mechanical allodynia and hyperalgesia via inhibi

Although vincristine is a frontline antineoplastic agent for treatment of tumors,the vincristine evoked pain syndrome is a serious problem for patients.There is currently no valid drug to prevent or t

会议

MgTallodyniaTNF-α

Sensitization of sodium channels contributes to colonic visceral hypersensitivity in diabetic rats a

Background:Intestinal dysfunction is a common chronic complication of diabetes mellitus,the underlying mechanisms of colonic visceral hypersensitivity in diabetic patients remain elusive.Recently,seve

会议

Diabetesvisceral hypersensitivitydorsal root ganglionvoltage-gated sodium cha

内吗啡肽-1后处理对大鼠心肌缺血再灌注损伤的影响及机制探讨

目的：观察内吗啡肽-1(Endomorphin-1,EM-1)后处理对心肌缺血再灌注损伤(myocardialischemia-reperfusion injury,IRI)的影响,并初步探讨EM-1 对I/R 可能的作用机制。方法：SD 雄性大鼠36 只,随机分为6 组：假手术组(S 组)、缺血再灌注组(IR 组)、缺血后处理组(IPO 组)、内吗啡肽-1 后处理组(50μg/kg)(EM50

会议

内吗啡肽-1缺血再灌注损伤缺血后处理大鼠

Inward rectifier K+ currents are regulated by CaMKⅡ in endothelial cells of primarily cultured bovin

Endothelium lines vascular walls and regulates vascular tones.The endothelial cells sense and respond to chemical and mechanical stimuli in the circulation,and couple the stimulus signals to vascular

会议

Inward rectifier K+currentsendothelial cellsCaMKⅡprimarily cultured bovine pu

Endogenous ET-1 regulates hypoxia-induced atrial ANP secretion via ETR-MAPK and ETR-Akt signaling pa

Endothelin-1 (ET-1) is produced in small amounts by the atrial endocardium and to control atrial natriuretic peptide (ANP) secretion.However,the effect of endogenous ET-1 on the regulation of hypoxia-

会议

ET-1ANPhypoxiaPI3K/AktMAPK-ERK

Contradictory role of apelin in vascular calcification

Apelin is a cardiovascular peptide with multiple functions regulating homeostasis of the circulatory system and is the endogenous ligand of angiotensin receptor AT1 (APJ).The role of apelin/APJ system

会议

vascular calcificationapelinAPJsmooth muscle cell

Flow-dependent Epigenetic Mechanism in Regulation of Vascular Endothelial Proliferation and Inflamma

Shear stress,the tangential component of the hemodynamic forces acting on the vascular wall,has been shown to play crucial roles in the regulation of vascular endothelial function,as well as the forma

会议

EndotheliumShear stressAtherosclerosisDNA methylationDNMT1

Cathepsin K effects Ischemia-Induced Neovascularization in old Mice

Cysteinyl cathepsins (Cats) have been implicated in the process of angiogenesis.we investigated the role of CatK in ischemia-induced neovascularization with a special focus on Notch1 activation in old

会议

Cathepsin KNeovascularizationIschemiaold

Endoplasmic reticulum oxidoreductin 1α mediates hepatic endoplasmic reticulum stress in homocysteine

Endoplasmic reticulum (ER) stress is emerging as an important modulator of different pathological process and as a mechanism contributing to homocysteine (Hcy)-induced hepar injury.However,the molecul

会议

endoplasmic reticulum stressendoplasmic reticulum oxidoreductin 1αhomocysteine

Foot shock potentiates blood coagulation system of rats

与本文相关的学术论文