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Objective Tyrosine kinase with immunoglobulin and epidermal growth factor homology domain-2 (Tie2) is involved in modulation of the function of endothelial progenitor cells (EPCs).Shear stress is an important non-pharmacologic regulation strategy of EPCs.It is not clear whether shear stress can enhance reendothelialization capacity of EPCs after arterial injury via activating Tie2-dependent signaling.