FXYD2, a γ subunit of Na+,K+-ATPase, maintains persistent mechanical allodynia induced by inflammati

来源 :The 6th Asian Pain Symposium, APS 2015(第六届亚洲疼痛研讨会) | 被引量 : 0次 | 上传用户：pangzhu311

【摘要】

：

　　Objective Na+,K+-ATPase (NKA) is required to generate the resting membrane potential in neurons.Our previous study showed that activation of NKA in dorsal r

【作者】

：

BingCai[1]FengWang[1]Kai-ChengLi[1]Xu-YeHu[2]Ying-JinLu[1]QiongWang[3]LanBao[4]XuZhang[5]

【机构】

：

Institute of Neuroscience and State Key Laboratory of Neuroscience, CAS Center for Excellence in Bra

【出处】

：

The 6th Asian Pain Symposium, APS 2015(第六届亚洲疼痛研讨会)

【发表日期】

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2015年期

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　　Objective Na+,K+-ATPase (NKA) is required to generate the resting membrane potential in neurons.Our previous study showed that activation of NKA in dorsal root ganglin (DRG) neurons by follistatin-like 1 (Fstl1) could inhibit neurotransmission and elevate threshold of somatosensation.

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FXYD2, a γ subunit of Na+,K+-ATPase, maintains persistent mechanical allodynia induced by inflammati

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