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Immunological response induced by Helicobacter pylori (Hp.) discussed as regards the interaction of the bacteria with the phagocytes and involvement of T cells.The cytokines released during the inflammation data as conducive to persistence Hp.infection and the evidence to a risk factor for gastric carcinoma are also presented.The structure of lipid A of Hp.is also involved in persistence of the infection.The LPS may regulate the expression of adhesions and it can diminish the secretion of inflammatory cytokines by the host cells.The LPS may regulate the expression of adhesions and it can diminish the secretion of inflammatory cytokines by the host cells.A negative correlation between Hp.LPS driven proliferation of mononuclear leukocytes isolated from dyspeptic children, and the gastric inflammation was shown in the own papers.