论文部分内容阅读
本实验利用运动性早期肌僵硬的动物模型对运动性早期肌僵硬时肌细胞内乳酸脱氢酶(LDH)和肌酸激酶(CK)的活性进行了测定。同时参考了血乳酸浓度肌电活动变化和肌肉收缩力变化,力求为探讨早期肌僵硬提供依据。结果显示,早期肌僵硬时,上述两种酶活性下降,血乳酸升高,出现肌肉自发肌电活动和收缩力下降。提示早期肌僵硬与运动时代谢物堆积、ATP 不足所致的内环境变化可能有较密切的关系。
In this study, the activity of lactate dehydrogenase (LDH) and creatine kinase (CK) in muscle cells during early exercise-induced muscle stiffness was determined by animal models of early exercise-induced muscle stiffness. At the same time, the change of myoelectric activity and the change of muscle contractility with reference to the concentration of blood lactate were investigated. The aim of this study was to provide basis for exploring the early muscle stiffness. The results show that early muscle stiffness, the two enzymes decreased activity, increased blood lactate, muscle spontaneous myoelectric activity and decreased contractility. It is suggested that there may be a close relationship between early muscular stiffness and accumulation of metabolites during exercise and changes in the internal environment caused by ATP deficiency.