在细胞水平上，本文从生物光子学的角度研究了激光的生物刺激作用，提出了激光生物刺激作用的生物信息模型（ＢＩＭＬ）。ＢＩＭＬ假设，细胞生色团对弱激光的吸收允许弱激光光子象激素一样成为生物信息的载体。对于细胞膜上的生色团，ＢＩＭＬ进一步假设红、橙、黄等暖色激光通过Ｇｉ蛋白活化ｃＡＭＰ的磷酸二酯酶，通过Ｇｇ蛋白活化磷酯酶Ｃ或活化蛋白激酶关联受体使胞内ｃＡＭＰ水平降低；绿、蓝、紫等冷色激光通过Ｇｓ蛋白活化腺苷酸环化酶使胞内ｃＡＭＰ水平升高。本文通过应用验证了ＢＩＭＬ。本文还从时间量子力学的角度研究了ＢＩＭＬ的合理性 At the cellular level, this paper studies the biostimulation of laser from the perspective of biological photonics, and proposes the bioinformatics model (BIML) of laser biostimulation. BIML assumes that the absorption of weak lasers by cytochromes allows weak laser photons to act as bioinformatics carriers, like hormones. For the chromophore on the cell membrane, BIML further assumes that red, orange, yellow and other warm-colored lasers activate cAMP phosphodiesterase through the Gi protein, activate phospholipase C via Gg protein or activate protein kinase-associated receptors to bring intracellular cAMP levels Decreased; green, blue, purple and other cool laser through Gs protein activation of adenylate cyclase intracellular cAMP levels. This article verifies BIML by application. In this paper, we also study the rationality of BIML from the perspective of time quantum mechanics