患者男,42岁。因腹胀7月,双下肢无力渐加重4月,于93年1月29日入院。 患者早在1980年体检发现HBsAg阳性,当时肝功能正常,无特殊不适,未治疗。1992年7月起感觉腹胀、尿少。“B”超检查提示:肝硬化腹水。在外院经用护肝、利尿及支持疗法,病情未缓解。92年10月出现双下肢无力,行走困难,渐加重。伴食欲不振,双上肢乏力。查体:被动体位,面色潮红,巩膜无黄染。心肺无特殊。腹部高度膨隆,肝、脾肋下触诊不满意,双下肢呈不完全性软瘫,肌力为Ⅲ级。四肢肌张力均低下,轻度肌萎缩。痛、温、触觉及深部感觉正常,双侧腱反射消失,未引出病理反射。实验室检查:血清钾6.28mmol/L,钠128.8mmol/L,氯102.4mmol/L,钙2.9mmol/L,谷—丙转氨酶 Male patient, 42 years old. July due to abdominal distension, weakness in both lower extremities gradually increased in April, January 29, 1993 admission. Patients as early as 1980 physical examination found HBsAg positive, normal liver function, no special discomfort, untreated. Since July 1992, I feel bloating and oliguria. “B” ultra-test prompts: cirrhosis of the ascites. In the out hospital with liver protection, diuretic and supportive therapy, the disease did not ease. 92 years in October double lower limb weakness, walking difficulties, gradually heavier. With loss of appetite, both upper extremity fatigue. Physical examination: passive position, flushing, sclera no yellow dye. No special cardiopulmonary. Abdominal height bulge, liver, spleen rib palpation are not satisfied, both lower extremities were incomplete soft paralysis, muscle strength of grade III. Limb muscle tension are low, mild muscle atrophy. Pain, temperature, touch and deep feeling normal, bilateral tendon reflex disappeared, did not lead to pathological reflex. Laboratory tests: serum potassium 6.28mmol / L, sodium 128.8mmol / L, chlorine 102.4mmol / L, calcium 2.9mmol / L, glutamyl transaminase