前列腺烷(PGs)是所有内脏合成的旁分泌物质,其中有舒张血管的PGI_2、PGE_2和收缩血管的血栓素B_2(TXB_2)。过去的研究提示在急性出血性休克时内脏血管床可代偿性地增加内源性PGI_2的合成和释放,但近期研究证实该代偿能力有限。为此作了进一步研究。取300g重的雄性S-D鼠,麻醉后行右颈动脉和股动脉插管,前者连接微机以连续监测动 Prostanoids (PGs) are paracrine materials for all visceral synthesis, including vasodilator PGI_2, PGE_2, and thromboxane B_2 (TXB_2) that constrict blood vessels. Past research suggests that the vasculogenic vascular bed compensates for the synthesis and release of endogenous PGI 2 during acute hemorrhagic shock, but recent studies have demonstrated limited compensatory capacity. To this end made a further study. Take 300g heavy male S-D rats, the right carotid artery and femoral artery anesthesia after intubation, the former connected to the computer to continuously monitor the move