3例藏族男性患者（例1，64岁；例2，51岁；例3，39岁）既往均有痛风病史，为行内镜下检查或治疗做肠道准备而服用磷酸钠盐口服溶液90 ml。服药后第2天均出现典型痛风发作，但血尿酸均比服药前下降（例1，526 μmol/L降至417 μmol/L；例2，566 μmol/L降至374 μmol/L；例3，465 μmol/L降至379 μmol/L）。考虑3例患者的痛风急性发作可能与磷酸钠盐口服溶液有关，是否与磷酸盐结晶相关尚不能确定。“,”Three Tibetan male patients (patient 1, 64-year-old; patient 2, 51-year-old; patient 3, 39-year-old) with previous history of gout received sodium phosphates oral solution 90 ml in intestinal preparation for endoscopic examination or treatment. The patients all developed typical gout attack on day 2 after their medications, but their blood uric acid levels were lower than those before taking the medicine (patient 1, from 526 μmol/L to 417 μmol/L; patient 2, from 566 μmol/L to 374 μmol/L; patient 3, from 465 μmol/L to 379 μmol/L), respectively. The acute attack of gout in the 3 patients was considered to be related with sodium phosphates oral solution, but whether it was related with phosphate crystallization was not determined.