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缺血后处理是保护心肌的一种新方法,其机制与抑制氧自由基堆积、抑制钙超载、抑制中性粒细胞的活化及激活再灌注损伤的补救激酶途径和ATP敏感性钾通道开放等有关。
Ischemic postconditioning is a new way to protect myocardium by inhibiting the accumulation of oxygen free radicals, inhibiting calcium overload, inhibiting the activation of neutrophils and activating the aortic kinase pathway and ATP-sensitive potassium channel opening in reperfusion injury related.