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全球多项研究显示,高尿酸血症是缺血性脑卒中的独立危险因素之一,血尿酸水平升高可增加脑卒中的发生率和死亡率。高尿酸血症与缺血性卒中的危险因素(高血压、糖尿病、高血脂、动脉粥样硬化等)共同促进缺血性脑卒中的发生。对高尿酸血症的治疗已经从急性期治疗转变为强调长期控制血尿酸达标,预防各种合并症。高尿酸血症的治疗包括调整饮食结构、改变生活方式、积极控制与血尿酸升高相关的危险因素、避免应用血尿酸升高的药物、碱化尿液等。降低血尿酸的药物包括增加尿酸排泄的药物、抑制尿酸合成药物、辅助降尿酸药。如果引起血尿酸增高的原因不能去除,需要长期降尿酸治疗。
A number of studies worldwide have shown that hyperuricemia is an independent risk factor for ischemic stroke. Elevated serum uric acid levels can increase the incidence of stroke and mortality. Hyperuricemia and ischemic stroke risk factors (hypertension, diabetes, hyperlipidemia, atherosclerosis, etc.) together to promote the occurrence of ischemic stroke. The treatment of hyperuricemia has changed from acute treatment to emphasize long-term control of blood uric acid compliance, prevention of various complications. The treatment of hyperuricemia includes adjusting the diet structure, changing the lifestyle, actively controlling the risk factors associated with the rise of serum uric acid, avoiding the use of drugs with elevated serum uric acid, alkalizing urine and the like. Lower blood uric acid drugs include increased uric acid excretion of drugs, inhibit uric acid synthesis drugs, adjuvant uric acid drugs. If the cause of increased blood uric acid can not be removed, the need for long-term uric acid reduction.