利用透射电镜对小麦白粉病菌与不同抗性寄主相互作用中乳突反应的超微结构进行了系统研究．结果表明，在超微结构上乳突反应是所有侵染位点的共有特征；线粒体、多聚核糖体、高尔基体及各种小囊泡参与了小麦乳突的形成；进入乳突沉积区的大量小囊泡首先形成一个个结构致密的小颗粒，然后堆积起来形成乳突的内部主体和核心，其外围是细胞器解体后沉积的一层染色淡而均匀的无定形物质．乳突沉积开始的早晚及其沉积速度和持续时间决定乳突最终的形态结构、大小及其抗性强弱．乳突抗性的决定因素是乳突沉积的早晚与速度，而不是最终沉积量．乳突抗性强的材料如ＫｈａｐｌｉｘＣｃ８总是在入侵栓进入前已形成较完整的半圆形乳突；而高感寄主如高加索的绝大部分乳突物质是在病菌入侵栓进入后沉积的，所形成的多是沿吸器颈部沉积的筒状无抗性乳突．病菌侵染时寄主细胞壁上过氧化物酶活性显著增强，但与乳突抗性无关 The ultrastructure of mastoid reaction in the interaction between wheat powdery mildew and different resistant hosts was investigated by transmission electron microscopy. The results showed that the mastoid reaction was a common feature at all sites of infection on the ultrastructure. Mitochondria, polysomes, Golgi bodies and various vesicles were involved in the formation of wheat papillae. A large number of small vesicles first formed a compact structure of small particles, and then piled up to form the internal body and core of the papilla, the periphery of the organelles deposited after deposition of a layer of light and uniform deposition of amorphous material. Mastosis deposition sooner or later and its deposition rate and duration of the final shape of the mastoid structure, size and strength of resistance. Determinants of mastoid resistance are the rate of morning and evening papilla deposition, not the final deposition. Mastoid-resistant materials such as KhaplixCc8 always form a more complete semicircular papilla prior to invasion of the plug; whereas most mastoid substances, such as Caucasus, which are highly susceptible hosts, are deposited after the invasion of the germ plug, Mostly formed are cylindrical unimpeded papillae deposited along the neck of the inhaler. Peripheral blood lipid peroxidase activity was significantly increased in host cells during infection, but not with mastoid resistance