这是因为矽尘作业工人吸入矽尘后,矽尘刺激肺组织引起肺内巨噬细胞的增生,巨噬细胞大量地吞噬矽尘,矽尘的溶解产物导致巨噬细胞的死亡、崩溃,释放出有毒的细胞液和残余的矽尘,引起肺组织的纤维化。如此反复地致纤维化作用,时间久了就会产生典型的矽肺病变。矽尘作业工人脱离矽尘作业时进行胸部X线照片,虽然未发现矽肺,但因肺内存在大量矽尘,上述病理反应过程仍不断地进行,故可于脱离矽尘作业若干年后还发生矽肺,这就叫做晚发性矽肺。 This is because silica dust inhalation of silica dust workers, silica dust stimulate lung tissue caused by the proliferation of macrophages in the lungs, macrophages phagocytose a large number of silica dust, silica dust lysate lead to the death of macrophages, collapse, release Toxic cell fluid and residual silica dust, causing lung tissue fibrosis. This fibrosis caused by repeated, over time will have a typical silicosis. Silica dust workers from the work of silica dust thoracic X-ray photographs, although no silicosis, but due to the presence of a large number of silicosis in the lungs, the pathological reaction process is still ongoing, it can be separated from the silica dust after a number of years of operation also occurred Silicosis, which is called late silicosis.