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电镜观察表明，锌酞菁人血清低密度脂蛋白（ＬＤＬ）复合物对小鼠ＭＳ－２纤维肉瘤细胞有很强的直接光敏杀伤作用，瘤细胞出现一系列结构改变：核染色质凝聚、局部核膜孔消失、核固缩、核破裂、凝聚的染色质流散、胞质内吞噬现象、胞膜表面肿胀粗钝的胞突形成、细胞碎裂等。提示该复合物光敏作用可诱导肿瘤细胞的程序性死亡，为解释其光敏杀伤机理提供了新的论据。
Electron microscopy showed that zinc phthalocyanine human serum low-density lipoprotein (LDL) complexes had a strong direct photosensitization effect on mouse MS-2 fibrosarcoma cells, and a series of structural changes appeared in the tumor cells: nuclear chromatin condensation, local The disappearance of nuclear pores, nuclear condensation, nuclear rupture, condensation of condensed chromatin, cytoplasmic phagocytosis, swelling of the cell membrane surface, dull formation of cells, cell fragmentation. It is suggested that the photosensitizing effect of the complex can induce the programmed cell death of tumor cells, which provides a new argument for explaining the mechanism of photodegeneration.