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已有研究显示，消化道的慢性炎症性疾病有发展成癌的可能，这与诱导性一氧化氮合酶（Induciblenitricoxidesynthase，iNOSorNOS２）高表达所导致NO过氧化损伤有一定关系犤１犦，但在无明显炎症性疾病的大肠癌发生发展中，NOS２变化及作用机制尚不十分清楚。本实验通过观察NOS２
It has been reported that chronic inflammatory diseases of the digestive tract may develop into cancerous cells, which is related to the NO-induced oxidative damage induced by the high expression of inducible nitric oxide synthase (iNOSorNOS2) In the development and progression of colorectal cancer without obvious inflammatory diseases, the mechanism of NOS2 changes and its mechanism is not yet clear. The experiment by observing NOS2