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非溃疡性消化不良 (ＮＵＤ)病人对机械扩张刺激的内脏感觉阈值明显下降[1 ,2 ] ,然而对ＮＵＤ病人内脏感觉过敏的机制却不甚了解。动物实验证实 :胃肠道Ｃ纤维末梢释放的降钙素基因相关肽 (ＣＧＲＰ)、Ｐ物质 (ＳＰ)对内脏感觉的产生及初级传入感觉纤维的致敏起着非常重要的作
Visceral sensory thresholds to mechanical dilation stimuli in patients with non-ulcerative dyspepsia (NUD) have been significantly reduced [1,2], yet mechanisms of visceral hypersensitivity in NUD patients are poorly understood. Animal experiments confirmed that: the release of calcitonin gene-related peptide (CGRP), substance P (SP) on visceral sensation and primary sensory fiber sensory fiber plays a very important role