论文部分内容阅读
在各种病因诱发急性肺损伤(ALI)/急性呼吸窘迫综合征(ARDS)的病生理过程中,受损肺泡及其基底膜的水肿增厚是直接导致氧和二氧化碳气体交换障碍的基础。钠泵——Na~+-K~+-ATPase活性可能会在其中有变化。本研究以肺组织湿,干重比(W/D)作为总肺水含量来探讨兔内毒素性急性肺损伤时其与肺组织Na~+-K~+-ATPase活性变化间的关
Thickening of damaged alveoli and their basement membrane edema is the direct cause of barriers to the exchange of oxygen and carbon dioxide gas in the pathophysiology of various causes of acute lung injury (ALI) / acute respiratory distress syndrome (ARDS). Sodium pump - Na ~ + -K ~ + -ATPase activity may change in them. In this study, lung wet and dry weight ratio (W / D) as the total lung water content to explore the endotoxin-induced acute lung injury in rabbits and lung tissue Na ~ + -K ~ + -ATPase activity between