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根据研究,实验性高血压在出现高血压症状的前期,已经有了血管平滑肌细胞膜离子转运功能异常,它可表现为在高盐负荷时对一些血管加压药物反应性异常增高。因此,可以利用肾上腺负荷实验(血管加压实验)确定血管平滑肌细胞膜离子转运功能状态,限制氯化物摄入量,予防高血压产生。国外资料介绍长期饮用氯化物1.4g/1(以C1-计)水的居民,能出现高血压症状或动脉压对肾上腺素负荷表现异常增高。大鼠长期(12个月)饮氯化物10g/1水能产生高血压症状,饮氯化物2.5g/1水虽引起高血压症状,但肾上腺素负荷时动脉收缩压
According to the study, there was an abnormal ion transport function of vascular smooth muscle cells in experimental hypertension at the early stage of symptoms of hypertension, which can be manifested as abnormally increased reactivity to some vasopressors under high salt load. Therefore, we can use the adrenal load test (vascular compression test) to determine the vascular smooth muscle cell membrane ion transport function, limit chloride intake, to prevent hypertension. Foreign information on long-term consumption of chloride 1.4g / 1 (C1- meter) water residents, there may be symptoms of hypertension or arterial pressure abnormalities in the performance of adrenaline increased. Rats long-term (12 months) drinking chloride 10g / 1 water can produce high blood pressure symptoms, chlorhexidine chloride 2.5g / 1 water caused by symptoms of hypertension, but arterial systolic pressure