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本文对31例正常人,75例高脂血症及53例冠心病患者口服100克葡萄糖后血糖及血浆胰岛素水平进行了分析。结果发现,高脂血症及冠心病患者(Ⅱb及Ⅳ2型)耐糖降低及血浆甘油三酯增高并非胰岛素分泌不足所致。相反,患者负荷糖后胰岛素的分泌较正常显著增加,峰值延后(60及120分钟);3小时后仍未恢复至空腹水平;胰岛素分泌面积增加;呈现高胰岛素血症。本文对高胰岛素血症的产生及其与血脂升高,耐糖降低及动脉硬化的关系进行了设想及讨论。
In this paper, 31 cases of normal subjects, 75 cases of hyperlipidemia and 53 cases of coronary heart disease in patients with oral glucose 100 grams of glucose and plasma insulin levels were analyzed. The results showed that patients with hyperlipidemia and coronary heart disease (Ⅱ b and Ⅳ type 2) decreased blood glucose and plasma triglyceride is not caused by inadequate insulin secretion. In contrast, the patient’s insulin secretion after loading significantly more than normal, peak delay (60 and 120 minutes); after 3 hours has not yet returned to fasting levels; insulin secretion increased; showing hyperinsulinemia. In this paper, the hyperinsulinemia and its relationship with elevated blood lipids, hypoglycemic and atherosclerosis were also discussed and discussed.