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脑出血后血肿周围存在继发炎性反应与自由基损伤。近几年来有些资料表明自由基损伤也参与脑出血后的继发损伤,而炎性反应与自由基损伤可能存在相互作用,通过多渠道、多环节,产生一系列级联式反应,加重脑出血后继发性损伤。文章从血肿周围组织炎性细胞浸润、炎性因子释放、补体激活几方面简单阐述血肿周围继发炎性反应损伤的机制。
There are secondary inflammatory reactions and free radical damage around the hematoma after intracerebral hemorrhage. In recent years, some data show that free radical damage is also involved in secondary injury after intracerebral hemorrhage, and there may be interaction between inflammatory reaction and free radical damage. Through multiple channels and multiple links, a series of cascade reactions occur, which aggravate cerebral hemorrhage After secondary injury. This article briefly describes the mechanism of secondary inflammatory reaction injury around the hematoma from the aspects of inflammatory cell infiltration, release of inflammatory cytokines and complement activation.