成人呼吸窘迫综合征(ARDS)和脓毒症引起的多器官衰竭(MOF)是创伤和腹膜炎病人最常见的死因。研究表明中性白细胞产生的氧自由基(OFR)对急性炎症起重要作用。有人断言,OFR引起的脓毒性休克成为脓毒症患者器官损害的原因。实验旨在用超氧化物歧化酶(SOD)和过氧化氢酶(CAT)对OFR的早期形成进行干预。并在对实验鼠使用SOD和CAT后,对临床效果、死亡率、类脂过氧化反应物硫 Adult respiratory distress syndrome (ARDS) and sepsis-induced multiple organ failure (MOF) are the most common causes of death in trauma and peritonitis. Studies have shown that oxygen free radicals (OFRs) produced by neutrophils play an important role in acute inflammation. It has been asserted that OFR-induced septic shock contributes to organ damage in septic patients. The experiment aimed to intervene the early formation of OFR with superoxide dismutase (SOD) and catalase (CAT). And in the use of SOD and CAT after the experiment, the clinical effect, mortality, lipid peroxidation of sulfur