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本实验采用以TP801微处理机为中心的“可编程刺激器”观察了急性不完全缺血心肌不应期、离散度及室颤阈的变化,探讨了室颤的机制。证实:1.不完全缺血心肌不应期呈现出多种变化,其中以不应期延长与缩短同时存在于缺血区为主,即使同一个点,不应期也可能出现延长与缩短交替出现的变化。2.心肌兴奋恢复离散度明显高于缺血前(p<0.01)。3.不完全缺血20分钟内左室室颤阈明显低于缺血前(P<0.01)。根据实验结果认为,急性心肌不完全缺血可能是发生室颤的一个重要原因。
In this experiment, TP801 microprocessor-centered “programmable stimulator” was used to observe the changes of refractory, discrete and ventricular fibrillation thresholds in acute incomplete ischemic myocardium. The mechanism of ventricular fibrillation was also discussed. Confirmed: 1. Incomplete ischemic myocardial refractory period showed a variety of changes, of which the refractory period and shortening simultaneously exist in the ischemic area, even if the same point, refractory period may also appear to extend and shorten the alternation Changes that have occurred. Myocardial excitability recovery dispersion was significantly higher than before ischemia (p <0.01). Left ventricular fibrillation threshold within 20 minutes after incomplete ischemia was significantly lower than before ischemia (P <0.01). According to the experimental results, incomplete myocardial ischemia may be an important cause of ventricular fibrillation.