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感染恶性疟原虫的红细胞粘附于静脉内皮(深部血管内裂体生殖)和毛细血管的阻塞(脑型疟疾),是由于感染恶性疟原虫的红细胞膜局部和全部的改变所致。深部血管内裂体生殖和脑型疟疾的机制,已根据对猕猴体内的P.coatneyi和夜猴体内的恶性疟原虫的电子显微镜观察以及对猕猴感染诺氏疟原虫的流变学研究进行了推论。至今,在疟疾研究中,虽然显示出不同程度的深部血管内裂体生殖,但在感染恶性疟原虫和P.coatneyi的红细胞膜上特有的结球突起是与明显的深
Erythrocytes infected with P. falciparum adhere to the endothelium of the veins (deep endoplasmic reticulum) and capillaries (brain-type malaria) due to local and total alteration of the erythrocyte membrane infected with P. falciparum. The mechanism of deep endovascular reproduction and cerebral malaria has been inferred based on electron microscopic observations of P. copenici in macaques and P. falciparum in a nocturnal monkey and on the rheology of rhesus macaques infected with rhesus macaques . To date, although malaria research shows varying degrees of deep endovascular reproduction, the distinct knobs on the erythrocyte membranes infected with P. falciparum and P. cohorti are markedly deep