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本文报道重症呼吸衰竭存活病儿102例中的36例随访情况。随访时距惠儿病愈出院时间为6月～4年。随访时年龄为7月～16岁。随访项目与结果:1.36例体格检查,2例脑性瘫痪,体格发育测量评价8例(24%)在同龄水平的第20百分位以下。2.智力测定,36例中10例(28%)智力落后。3.肺部X 线摄片,33例中21例(64%)存在程度不同的肺部损害(肺间质改变、肺纤维化、肺不张)。4.脑干听力诱发电位测定,26例中10例(38%)异常。5.肺功能检查:3例呼气峰值气流均低于正常,2例用力肺活量降低。6.脑电图及脑电地形图,检查4例,3例异常。本文根据随访结果探讨其原因为综合因素,如原发病低氧血症致缺氧性脑病、较高浓度氧持续吸入及人工呼吸器机械损害等均为导致后遗症的原因。同时提出了改进治疗措施。
This article reports 36 cases of 102 survivors with severe respiratory failure who were followed up. From the benefits of pediatric patients were discharged from the hospital for 6 months to 4 years. At follow-up, the age was from July to 16 years old. Follow-up projects and results: 1.36 cases of physical examination, 2 cases of cerebral palsy, physical development measurement evaluation of 8 cases (24%) below the 20th percentile of the same age level. 2. Intelligence test, 36 cases in 10 cases (28%) mental retardation. 3. Pulmonary X-ray, there were 21 cases (64%) of 33 cases with different degrees of lung damage (interstitial lung fibrosis, atelectasis). Brainstem auditory evoked potentials, 26 cases, 10 cases (38%) anomalies. Pulmonary function tests: 3 cases of peak expiratory flow were lower than normal, 2 cases of forced vital capacity decreased. 6. EEG and EEG topography, check 4 cases, 3 cases of abnormalities. This article based on the follow-up results to explore its causes for a combination of factors, such as the primary hypoxemia-induced hypoxic encephalopathy, continuous inhalation of higher concentrations of oxygen and mechanical damage caused by respirators are the causes of sequelae. At the same time proposed to improve treatment.