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许多实验研究表明,脂质过氧化可导致肺巨噬细胞生物膜的损伤,参与粉尘所致肺组织纤维化形成的病理过程。另有实验研究认为水泥尘对肺巨噬细胞具有毒性作用。为探讨水泥生产工人机体内脂质过氧化及其生物学意义,我们对这些工人血中过氧化脂质(LPO)和超氧化物歧化酶(SOD)水平进行了测
Many experimental studies have shown that lipid peroxidation can lead to the injury of lung macrophage biofilm and participate in the pathological process of lung fibrosis caused by dust. Another experimental study that cement dust on lung macrophages has toxic effects. To investigate lipid peroxidation and its biological significance in cement production workers, we measured the level of blood lipid peroxidation (LPO) and superoxide dismutase (SOD) in these workers