【摘 要】
：
妊高征病因学说众多,其病理变化机制多认为血管内皮损伤和滋养层浸润障碍是妊高征发生过程中的两个主要环节[1]。近年来,临床上该类患者有逐年增加的趋势,但特异的实验室诊断
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妊高征病因学说众多,其病理变化机制多认为血管内皮损伤和滋养层浸润障碍是妊高征发生过程中的两个主要环节[1]。近年来,临床上该类患者有逐年增加的趋势,但特异的实验室诊断指标报道很少,本文试检测59例妊高征患者血浆凝血酶调节蛋白(TM)、超敏C反应蛋白(hs-CRP)和血清可溶性血管内皮细
The pathogenesis of PIH is numerous, and its pathological mechanism of many think that vascular endothelial injury and trophoblast infiltration disorders are two major links in the process of PIH [1]. In recent years, the clinical trend of these patients have increased year by year, but the specific laboratory diagnostic indicators rarely reported in this paper test 59 cases of PIH plasma thrombomodulin (TM), high-sensitivity C-reactive protein -CRP) and serum soluble vascular endothelial cells
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