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为探讨蛇床子水煎剂的抗氧化机制，本实验采用D－半乳糖衰老小鼠，灌喂蛇床子水煎剂，分别于15，30，45d处死，测定小鼠脑和肝组织谷胱甘肽过氧化物酶（GSH－Px）丙二醛（MDA）的活性．实验结果表明：与D-半乳糖衰老模型组比较，蛇床子水煎剂能明显提高衰老小鼠脑和肝的GSH－Px活性，明显降低MDA的含量．提示蛇库于水煎剂对衰老小鼠有抗氧化作用，可延缓小鼠脑和肝的衰老．
In order to investigate the anti-oxidation mechanism of Cnidium decoction, this experiment used D-galactose-sensing mice and was fed with Cnidium decoction, and were sacrificed at 15, 30, and 45 d, respectively. The brain and liver tissues of mice were measured for glutathione. Peptide Peroxidase (GSH-Px) Malondialdehyde (MDA) activity. The experimental results show that compared with the D-galactose aging model group, Cnidium decoction can significantly increase the activity of GSH-Px in the brain and liver of aging mice, and significantly reduce the content of MDA. It is suggested that snake decoction has anti-oxidation effect on aging mice and can delay the aging of mouse brain and liver.