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药物防治声损伤或噪声性耳聋的研究已进行多年,并取得了一定的进展;但是仍处在继续研究阶段。高压氧防治声损伤的实验研究也是为解决这一课题进行的探索工作。据报道,柯蒂氏器官代谢旺盛,耗氧量高,对缺氧敏感。强声刺激时耳蜗血管收缩,听力下降。由此可以推想,声损伤或噪声性耳聋可能是由于强声引起柯蒂氏器官局部缺氧,造成毛细胞以及耳蜗组织代谢障碍的结果。鉴于高压氧治疗突发性耳聋有一定的疗效,吸氧减轻暂时性耳聋的报道,我们设想如能吸入适量的高压氧,可能对高强度声音造成的声损伤有预防或治疗作用。
Studies on the prevention and treatment of acoustic injury or noise-induced deafness have been underway for many years and some progress has been made; however, they are still in the research phase. Experimental research on the prevention and treatment of acoustic damage by hyperbaric oxygen is also an exploration work to solve this issue. It is reported that Corti’s organ metabolism, high oxygen consumption, sensitive to hypoxia. Strong stimuli cochlear vasoconstriction, hearing loss. It can be speculated that acoustic damage or noise deafness may be due to strong sound caused by partial oxic local organitis, causing hair cell and cochlear tissue metabolism disorders results. In view of hyperbaric oxygen treatment of sudden deafness have a certain effect, oxygen reduction temporary deafness reported, we assume that if inhaled the right amount of hyperbaric oxygen may cause sound damage caused by high-intensity sound prevention or treatment.