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在实验性高血压以及人体高血压中均显示活性氧生成明显增多,血管中通过NADP(H)氧化酶以及NOS解偶联而产生的活性氧(ROS)参与了高血压过程中血管功能的损害及血管的炎性反应。运用抗氧化治疗有利于减小对血管的损伤,降低高血压生成的终末器官损害。
Both reactive oxygen species (ROS), produced by NADP (H) oxidase and uncoupling of NOS in blood vessels, have been shown to be involved in the detriment of vascular function during hypertension both in experimental hypertension and in human hypertension And vascular inflammatory response. The use of anti-oxidative therapy is beneficial to reduce the damage to blood vessels and reduce the damage of end organ caused by hypertension.