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作者根据脑电图(EEG),胎心产力图(CTG)和酸硷平衡三者变化作为产时监护胎儿的方法,企图找出:胎儿缺氧酸中毒时,胎儿 EEG 变化的特点;EEG 的改变是否先于 CTG;EEG 能否对胎儿严重缺氧及酸中毒所致脑损伤提供有价值的信息。65例足月产妇分为无缺氧组(33例),及缺氧组(32例),神经系统正常婴儿必须具备:1分钟Apgar 评分>8分,5分钟为10分,脐动脉血 pH>7.20,剩余硷>15.8mEq/l。若有一项不符合上述条件即划为缺氧组。当宫口开3～5cm 时,破膜取毛细血管血 pH,继将特制的 EEG 及 CTG电极粘在胎头上,每10分钟监测一次。分娩后取脐动脉血化验,与所获得的 EEG、CTG 结果经统计学处理后加以比较。
Based on the changes of EEG, CTG and pH balance, the author attempts to find out the changes of fetal EEG during fetal hypoxia acidosis. EEG Whether changes preceded CTG; whether EEG could provide valuable information on fetal hypoxia and brain damage caused by acidosis. 65 cases of full-term mothers were divided into no hypoxia group (33 cases) and hypoxia group (32 cases). Neonatal normal infants must have Apgar score> 8 for 1 minute and 10 for 5 minutes. Umbilical arterial blood pH > 7.20, the remaining alkali> 15.8mEq / l. If there is a non-compliance with the above conditions are classified as hypoxia group. When the cervix open 3 ~ 5cm, take the capillary blood pH, followed by the special EEG and CTG electrodes stuck to the fetal head, monitoring once every 10 minutes. Umbilical arterial blood samples were taken after delivery, and compared with the EEG and CTG results obtained after statistical analysis.