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氧自由基可能参与低血容量性及感染性休克的病理过程。氧自由基损伤细胞膜及超微结构,影响血管内皮功能,参与再灌注损伤及多器官衰竭,促进休克向不可逆方向发展。实验性抗氧自由基治疗己有可喜苗头,这一领域的深入研究可能为低血容量性及感染性休克的治疗开辟新径。
Oxygen free radicals may participate in the pathogenesis of hypovolemic and septic shock. Oxygen free radicals damage the cell membrane and ultrastructure, affecting vascular endothelial function, involved in reperfusion injury and multiple organ failure, and promote the shock to the irreversible direction. Experimental antioxidant free radical therapy has gratifying signs, in-depth study in this area may open a new path for the treatment of hypovolemic and septic shock.