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本实验通过观察摄入ＡＦＢ１及Ｇｅ－１３２后Ｗｉｓｔａｒ大鼠和昆明小鼠体内ＳＯＤ活力的变化，进一步了解ＡＦＢ１代谢途径和Ｇｅ－１３２与自由基的关系。实验表明，摄入ＡＦＢ１的ＳＯＤ活力受到抑制，而摄入Ｇｅ－１３２后ＳＯＤ活力得到提高，且一次性摄入ＡＦＢ１后ＳＯＤ活力最终得到恢复，其中大鼠对ＡＦＢ１敏感性相对较大。
In this experiment, we observed the changes of SOD activity in Wistar rats and Kunming mice after ingesting AFB1 and Ge-132 to further understand the relationship between AFB1 metabolic pathway and Ge-132 and free radicals. Experiments showed that the SOD activity of AFB1 ingested was inhibited, but the activity of SOD was increased after Ge-132 ingestion, and SOD activity was finally restored after one-time ingestion of AFB1. Rats were more sensitive to AFB1.