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患者男性68岁,住院号1432,曾于1973年6月北京医学院三附院诊断为急性下壁及正后壁心肌梗塞;出院后多次复查为陈旧性下壁并正后壁心肌梗塞。此次饮酒后心绞痛发作持续4小时,于1984年3月24日下午4时收入我院,当时神智清楚,血压18.7/12.0kPa,即描绘心电图与过去比较大致相同,但患者仍有心前区压榨性疼痛,出冷汗,3月25日晚血压16.0/9.3 kPa,晚10时至翌晨8时ECG表现V_2-V_4 S-T段升高伴T波后支倒置,V_2导联R波振幅明显降低,未见Q波。从1984年3月26日ECG图与1981年11月17日ECG图比较心前导联R波振幅降低,以R V_2振幅降低最明显,V_1-V_3导联仍无Q波,但在V_4-V_6导联
The patient male 68 years old, hospital number 1432, was diagnosed with acute inferior wall and posterior wall myocardial infarction in the Third Affiliated Hospital of Beijing Medical College in June 1973; repeatedly discharged after the hospital was found to be old inferior wall and posterior wall myocardial infarction. The episode of angina pectoris continued for 4 hours, at 4 pm on March 24, 1984 income in our hospital, when the clear mind, blood pressure 18.7 / 12.0kPa, which depicts the ECG in the past is more or less the same, but patients still have preclinical crushing Pain, cold and sweat, blood pressure of 16.0 / 9.3 kPa on the evening of March 25, ECG of the V_2-V_4 ST segment elevated with T wave in the evening from 10 am to 8 am the next day, the amplitude of R wave in V 2 lead decreased significantly, No Q waves. From the ECG on March 26, 1984 to the ECG on November 17, 1981, the amplitude of R wave decreased compared with the ECG on November 17, 1981, and the amplitude of R V_2 decreased most obviously. However, there was still no Q wave in the V_1 -V_3 lead, Lead