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目的：为进一步探讨高原肺水肿的发病机理，为预防和治疗提供理论依据；方法：对进驻特高海拔地区的32名青年在上高原前（海拔1400m）和急进（4天）海拔5400m第3、8、20天分别拍摄后前往X线胸片，测量心肺12项指标并进行自身对照分析；结果：急进海拔5400m第3～20天内，心脏横径、心脏长径、心脏面积逐渐缩小；肺动脉干横径、肺动脉段突度、右肺下动脉横径、升主动脉横径、主动脉结横径、降支与气管比值逐渐增大；胸宽径和肺面积在第3天及第8天时缩小非常明显（P＜0．001及P＜0．01），在第20天基本习服后较上高原前稍有增大（P＞0．05）；结论：人体进入特高海拔地区第3～8天由于心脏代偿性排出增高及肺部反应性瘀血使两肺明显缩小，此阶段是高原肺水肿发病的高峰期。
Objective: To investigate the pathogenesis of high altitude pulmonary edema and provide a theoretical basis for prevention and treatment.METHODS: Thirty-two youths at the altitude of 1400m above sea level and 4m above sea level at 5400m 3 , 8, 20 days after shooting to X-ray, measurement of 12 indicators of cardiopulmonary and self-control analysis; Results: rapid advance 5400m 3 to 20 days, the heart diameter, long diameter heart, the heart area gradually reduced; pulmonary artery Dry transverse diameter, pulmonary artery segment, right inferior pulmonary artery diameter, ascending aortic diameter, aortic node diameter, descending branch and tracheal ratio gradually increased; chest width and lung area on the 3rd and 8th (P <0.001 and P <0.01), and slightly increased on the 20th day compared with that before the plateau (P> 0.05). CONCLUSION: The human body enters the extra-high altitude area On the 3rd to 8th days, both lungs were significantly reduced due to compensatory discharge of the heart and reactive blood stasis in the lungs. This stage is the peak of the incidence of altitude pulmonary edema.