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一般认为妊娠晚期胎儿尿液是羊水的主要来源,主要靠胎儿的吞咽和排泄来维持平衡。胎儿泌尿系畸形可产生羊水过少,其次,稽留流产或胎死宫内可因胎儿无尿而原有羊水又被重吸收,形成慢性的羊水丢失、羊水过少。第一位证实羊水过少时伴有羊膜病变者建议用“羊膜结节(Amnion nodosum)”一词命名该病症。其后许多学者也观察到羊水过少时有羊膜结节,并都认为此结节是继发于羊水量的减少,而不是羊水少的原因。国内有关这方面的报导尚未见到。我院自1955年至1984年共有婴检527例,在其中18例有泌尿系畸形的婴检病例中2例有羊膜结节
Generally believed that the third trimester of fetal urine is the main source of amniotic fluid, mainly by the swallowing and excretion of the fetus to maintain balance. Fetal urinary tract abnormalities can produce oligohydramnios, followed by missed abortion or fetal death can be due to fetal anuria and the original amniotic fluid has been reabsorbed to form a chronic loss of amniotic fluid, oligohydramnios. The first person to confirm amniotic disease with oligohydramnios suggests using the term “Amnion nodosum”. Since then, many scholars have also observed amniotic nodules when oligohydramnios, and that this nodule is secondary to the reduction of amniotic fluid volume, rather than less amniotic fluid reasons. Domestic reports on this area have not yet been seen. In our hospital from 1955 to 1984, a total of 527 cases of infant examination, in which 18 cases of urinary tract malformations in 2 cases of amniotic nodules