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本文检测67例脾虚证胃病患者胃粘膜超微结构、肠化生亚型、DNA、cAMP、微量元素及其氧化物如~3H TdR LcT,发现不完全性结肠型肠化生率、非病灶处“背景病变”发生率随着脾气虚证至脾虚气滞证、良性胃病至胃癌的顺序递增.组间均有显著性差异.P<0.05～0.001。胃粘膜不完全性结肠型肠化生组织内DNA、cAMP、Zn、Cu、ZnO与CuO含量与胃癌组织间无显著性差异。认为脾虚气滞证胃病伴不完全性结肠型肠化生,及cAMP、Zn、Cu、ZnO与CuO含量降低,而DNA含量异常增高时,有癌变倾向。
This article detected 67 cases of spleen deficiency stomach syndrome patients with gastric mucosal ultrastructure, intestinal metaplasia, DNA, cAMP, trace elements and their oxides such as ~ 3H TdR LcT, found incomplete intestinal metaplasia rate, non-lesion The incidence of “background lesions” increased with the order of spleen-qi deficiency syndrome, spleen deficiency qi stagnation syndrome, and benign gastropathy to gastric cancer. There was a significant difference between the groups. P <0.05-0.001. There was no significant difference in the contents of DNA, cAMP, Zn, Cu, ZnO, and CuO between gastric intestine and incomplete intestinal metaplasia of gastric mucosa. It is believed that spleen qi stagnation stomach syndrome with incomplete colonic intestinal metaplasia, and cAMP, Zn, Cu, ZnO and CuO content decreased, and DNA content increased abnormally, there is a tendency to cancer.