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匍枝根霉（Ｒｈｉｚｏｐｕｓｓｔｏｌｏｎｉｆｅｒ）和半裸镰刀菌（Ｆｕｓａｒｉｕｍｓｅｍｉｔｅｃｔｕｍ）以不同的机制侵染甜瓜果实。匍枝根霉侵染时，菌丝分泌大量的果胶甲酯酶（ＰＭＥ）、多聚半乳糖醛酸酶（ＰＧ）、果胶甲基半乳糖醛酸酶（ＰＭＧ），迅速消解组织中胶层，引起细胞电解质外渗、质壁分离和软腐。菌丝在胞外和细胞间隙生长，不能穿透细胞壁。半裸镰刀菌侵染甜瓜果实时分泌高活力的果胶甲酯酶、果胶裂解酶类（ＰＭＬ）和纤维素酶（Ｃｅｌｕｌａｓｅ）而缺少果胶水解酶类，菌丝不能迅速消解中胶层，但以直接穿透细胞壁方式侵染组织细胞。这两种病原对甜瓜不同的致腐方式是由它们不同的外泌胞壁降解酶种类和酶学特性所决定的。
Rhizopus stolonifer and Fusarium semitectum infest melon fruits with different mechanisms. Rhizopus stolonifer infection, mycelium secretion of large amounts of pectin methylesterase (PME), polygalacturonase (PG), pectin methyl galacturonase (PMG), rapid digestion of tissue Adhesive layer, causing cell electrolyte extravasation, plasmolysis and soft rot. Mycelium grows in the extracellular and intercellular spaces and can not penetrate the cell wall. Fusarium solani infects muskmelon fruits and secrete high activity of pectin methylesterase, pectin lyase (PML) and cellulase (Celulase) and lack of pectolytic enzymes, mycelium can not quickly digest the adhesive layer, However, the direct penetration of cell wall infection of tissue cells. The different pathogenic mechanisms of these two pathogens to melon are determined by their different exocellular cell wall degrading enzymes and enzymatic properties.