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较多实验研究表明,CCK在急性胰腺炎发生发展过程中具参与和促进作用,其机理可能在于干扰胰酶的外分泌。CCK受体阻滞剂对实验性急性胰腺炎有较好的防治作用。
More experimental studies have shown that, CCK in the development and progression of acute pancreatitis with participation and promotion, the mechanism may interfere with the secretion of pancreatic exocrine. CCK receptor blockers have a better preventive and therapeutic effect on experimental acute pancreatitis.