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假定患者碘的摄入量正常,那么蛋白结合碘升高,胆固醇降低,说明患者主要疾病是甲状腺机能亢进(以下简称甲亢),当患者发生心力衰竭时,甲亢变得更明显。患者血压的升高主要是收缩压升高。虽然脉搏较快,但舒张压无明显升高。这些临床资料表明,心搏出量和心输出量升高是代谢升高的一种表现。甲状腺素具有增强全身氧化代谢的作用,使组织舒血管代谢产物增加,从而使外周血管扩张,使血流量和静脉回流血量增多。这种高的心输出量的维持是靠代谢升高刺激血流量增加,以及甲状腺素增强交感神经对心脏的刺激作用,二者联合作用的结果。
Assuming normal intake of iodine in patients, elevated protein-bound iodine and reduced cholesterol indicate that the major disease in patients is hyperthyroidism (Hyperthyroidism), and when the patient develops heart failure, hyperthyroidism becomes more pronounced. Patients with elevated blood pressure is mainly systolic blood pressure. Although the pulse faster, but no significant increase in diastolic blood pressure. These clinical data show that cardiac output and cardiac output is elevated metabolic performance of a manifestation. Thyroxine can enhance the body’s role in oxidative metabolism, so that vascular tissue metabolites increased, so that peripheral blood vessels to expand, so that blood flow and venous return increased blood volume. This high cardiac output is maintained by increased metabolism to stimulate blood flow increases, and thyroid hormone enhances the sympathetic heart stimulation, the combined effect of the two.