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本例是由于心室电极脱位导致起搏器功能障碍。心房和心室电极脱位的迹象是起搏器诱发的室性早搏、导管诱发的短暂性室性心动过速和心室电极夺获心室失灵。当心房活动被充分感知的同时,心室夺获突然丧失而发生晕厥。附图A系心脏骤停前大约6.5小时所描记,心房活动60～75次/分。↑所示三次搏动的正常房室顺序起搏,心室起搏紧随患者自身窦性P波之后,系正常的VDI型(心室起搏和房室双腔感知)。起搏器功能障碍表现为:(1)紧随起搏P波之后的起搏脉冲没有夺获心室,见所示;(2)不伴起搏脉冲信号发生室性心动过速
This case is due to ventricular electrode dislocation lead to pacemaker dysfunction. Signs of atrial and ventricular electrode dislocations are pacemaker induced premature ventricular contractions, catheter-induced transient ventricular tachycardia, and ventricular electrodes to capture ventricular failure. When the atrial activity is fully perceived at the same time, the sudden loss of ventricular seizure occurred syncope. Figure A is about 6.5 hours prior to cardiac arrest, atrial activity of 60 to 75 beats / min. ↑ The normal beating sequence of three beats is shown in Figure 3. Ventricular pacing follows the patient’s own sinus P wave and is of normal VDI type (ventricular pacing and atrioventricular duplex sensing). Pacemaker dysfunction manifested as: (1) no pacing was taken of the pacing pulse immediately following pacing P wave, as shown; (2) ventricular tachycardia without pacing pulse signal