论文部分内容阅读
观察了大鼠力竭运动后心肌线粒体脂质过氧化水平、游离钙含量和磷服酶Ａ_２活性的变化，发现运动后即刻心肌线粒体脂质过氧化水平和磷脂酶Ａ_２活性显著升高，游离钙含量显著下降。结果提示力竭运动时心肌线粒体内源自由基的产生是心肌损伤和运动性疲劳的原因之一。
The changes of myocardial mitochondrial lipid peroxidation, free calcium and phospholipase A 2 activity after exhaustive exercise were observed. The myocardial mitochondrial lipid peroxidation and phospholipase A 2 activity were significantly increased after exercise, and the content of free calcium Significantly decreased. The results suggest that the production of endogenous free radicals in myocardial mitochondria during exhaustive exercise is one of the causes of myocardial injury and exercise-induced fatigue.