许多资料表明,体内钠蓄积与高血压发病密切相关。低钠饮食或利钠药都能降压;但对钠诱发高血压的机理,始终不清楚。Parini等研究了对钠盐敏感(SBH)的Sabra大鼠和耐钠盐(SBN)的 Sabra 大鼠在高钠饮食后,中枢皮层、下丘脑和延脑的α受体密度以及去甲肾上腺素含量的改变。SBN 大鼠和 SBH 大鼠分为二组,分别摄入正常钠(0.2%NaCl)与高钠(0.8%NaCl)。5周后, Many data show that in vivo sodium accumulation and the incidence of hypertension are closely related. Low-sodium diet or sodium can reduce blood pressure; but the mechanism of sodium-induced hypertension is still not clear. Parini et al. Studied Sabra rats with sodium sensitivity (SBH) and Sabra rats with sodium resistance (SBN) after a high sodium diet. The concentrations of alpha receptors in the central cortex, hypothalamus and medulla oblongata and norepinephrine Changes in content. SBN rats and SBH rats were divided into two groups, normal sodium (0.2% NaCl) and high sodium (0.8% NaCl) respectively. After 5 weeks,