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病毒性心肌炎(VM)是由于病毒侵犯心肌,在心肌细胞内复制而导致心肌溶解并由此诱发一系列心肌自身免疫反应的结果,其病理特征为心肌细胞的坏死或变性。VM的发病机制尚不完全清楚,但随着分子病毒学、分子免疫学的发展,揭示出VM发病机制涉及到病毒对感染的心肌细胞直接损害和病毒
Viral myocarditis (VM) is a result of a series of myocardial autoimmune reactions that result from viral invasion of the myocardium, replication within the cardiomyocytes leading to the dissolution of the myocardium and its pathological characteristics of necrosis or degeneration of the cardiomyocytes. The pathogenesis of VM is not fully understood, but with the development of molecular virology and molecular immunology, it is revealed that the pathogenesis of VM involves the direct damage of virus to infected cardiomyocytes and virus