不少文献都赞同这样的一种理论,即特异性的补体成份可以激活炎症细胞或效应细胞,使它们能直接或间接地去杀伤肿瘤细胞。为验证这一理论,本文作者意图用血清治疗的方法观察肿瘤部位的急性或慢性组织学改变。实验所用动物为雌性Buffalo/N大鼠,以N-亚硝基-n-甲基脲(NMU)诱发乳腺癌。所用血清由心脏直接取血,并经蛋白A-琼脂糖吸收处理。实验结果表明,经使用蛋白A-琼脂糖吸收的同基因血清后3～4小时,弥散的炎症细胞可浸润至大鼠原发性乳腺癌组织之 Many literatures agree with the theory that specific complement components can activate inflammatory or effector cells, allowing them to directly or indirectly kill tumor cells. To verify this theory, the authors intended to use serum therapy to observe acute or chronic histological changes in the tumor site. Animals used in the experiment were female Buffalo/N rats and breast cancer was induced with N-nitroso-n-methylurea (NMU). The serum used was taken directly from the heart and absorbed by Protein A-Sepharose. The experimental results show that diffuse inflammatory cells can infiltrate into rat primary breast cancer tissues after 3 to 4 hours of syngeneic serum absorption using protein A-agarose.