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糖尿病患者患冠心病的风险是非糖尿病的2~6倍。其主要发病机制包括:氧化应激水平增高,晚期糖基化终末产物的形成,蛋白激酶C的激活。对这些机制的阐述有着非常现实的意义,并将有助于研制开发治疗糖尿病心血管并发症的新型药物。
Diabetes patients with coronary heart disease risk is non-diabetic 2 to 6 times. Its main pathogenesis include: increased oxidative stress, the formation of advanced glycation end products, protein kinase C activation. The elaboration of these mechanisms has a very realistic meaning and will help to develop new drugs for the treatment of diabetic cardiovascular complications.