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肝硬化病人常常表现某些凝血因子合成减少、分解代谢增强或数量改变。本文作者于有潜在消耗性凝血病的肝硬化病人,皮下注射肝素钙,研究其改善血凝因子数值的意义。本组30例男性肝硬化病人中13例处于失代偿状态,至少具备以下几项中的2项(按 Franco 等1977年修订标准):①腹水;②肝源性脑病;③凝血酶原试验(Quick)<40%;④血清白蛋白<30g/L;⑤血清胆红素>68.4μmol/L。每个病例均于治疗前和用药后第7和第15天采集血标本。全部病例显示如下基础凝血象:纤维蛋白(原)裂解产物(FDP)值增加,
Patients with cirrhosis often show reduced synthesis of certain clotting factors, increased catabolism, or altered numbers. The authors in patients with potential consumptive coagulopathy of liver cirrhosis, subcutaneous injection of heparin calcium, to study the significance of improving the value of clotting factor. The group of 30 cases of male patients with cirrhosis in 13 cases of decompensation, with at least two of the following (according to Franco and other revised standards in 1977): ① ascites; ② Hepatic encephalopathy; ③ prothrombin test (Quick) <40%; ④ serum albumin <30g / L; ⑤ serum bilirubin> 68.4μmol / L. Blood samples were collected in each case before treatment and on days 7 and 15 after treatment. All cases showed the following basis of coagulation: increased fibrin (original) cleavage product (FDP)