一、矽结节是一个从生理反应过渡到病理阶段的产物当矽尘进入肺泡后,即引起肺泡壁Ⅱ型细胞的增殖,分泌出许多表面活性物质,激发几方面的巨噬细胞云集肺泡,排除矽尘。巨噬细胞吞噬矽尘后成为吞噬细胞,亦称尘细胞,利用其阿米巴运动,大部分通过肺泡囊、肺泡管到呼吸细支气管、终末支气管进入各级支气管,利用分泌液和纤毛运动向上移动,达到咽喉排出,成为痰液吐出,或咽入食道进入消化道,另一小部分则通过肺泡壁进入肺间质淋巴液中,经过淋巴系统向肺门淋巴结转移。在这个 First, the silicon nodules is a transition from the physiological reaction to the pathological stage of the product when silica dust into the alveoli, which causes alveolar wall type II cell proliferation, secretion of many surface-active substances, stimulate several aspects of macrophages gathered alveoli, Exclude silica dust. Phagocytic macrophages phagocytosis after becoming phagocytes, also known as dust cells, the use of its amoebic motility, most of the alveolar sac through the alveolar tube to the respiratory bronchioles, bronchial bronchial tubes into the terminal at all levels, the use of secretions and ciliary movement Move up to the throat discharge, as sputum spit, or pharyngeal esophagus into the digestive tract, the other a small part of the pulmonary interstitial lymph through the alveolar wall into the lymph through the lymphatic system to the hilar lymph node metastasis. at this